Current Status
Let me shed some light on a
few of the questions and/or
statements that have been
made to the AMAA office.
Do both parents need to be
carrier’s in order for the
calf to be affected?
Yes, PHA is a result of
simple autosomal recessive
trait. Even though the sire
and dam are normal in their
appearance, they can pass on
the genetic defect to their
offspring.
Genotype Terminology
Phenotype
PP PHA-free normal
Pp PHA-carrier normal
pp PHA-affected affected
1. Free parent mated to a
carrier parent. Resulting
progeny has a 50 percent
chance of being free or a 50
percent chance of being a
carrier.
2. Mating two carrier
parents. Resulting progeny
have a 25 percent chance of
being free, 50 percent
chance of being a carrier or
a 25 percent chance of being
affected.

How prevalent is this
genetic defect in the
Maine-Anjou population?
At this point, we simply do
not know. Rumor is far worse
than fact and we do not yet
have enough facts to answer
this question. One must
remember that all
descendents of a designated
carrier genetic line will
not necessarily be carriers.
(Refer to the tables above)
When will a commercial test
for PHA be available?
Dr. Beever gave a report to
the board at the Fort Worth
meetings on the progress
being made in locating a
gene marker for PHA. At this
time, there are still a few
pieces to the puzzle
missing. The timing does not
look favorable to have a
commercial test available by
the early part of the spring
breeding season. With some
luck, a commercial test may
be available by late spring
or early summer.
If we
do not have a commercial
test, when will a list of
carrier A.I. sires be
available?
Remember that two confirmed
cases by Dr. Steffen or Dr.
Beever that are parent
verified are needed in order
to call the parent or
parents a carrier. After
deemed a carrier, the owners
have the notification and
appeal process that can be
exercised. Without the
commercial test,
realistically only a few A.I.
sires may be deemed carriers
by spring breeding season.
How do we keep up with
updated listings?
AMAA will be providing a
link to our Web site,
www.maine-anjou.org
dealing with PHA.
This will be kept current
with any new PHA
developments.
Why has it taken this long
to explain to the membership
what PHA is all about?
At the onset, June 2005, the
AMAA was just introduced to
the potential genetic defect
problem. An immediate
research program was set in
motion. Notification was
sent by letter to the entire
membership in September 2005
with an introductory article
in the October 2005 issue of
the Voice. As facts were
gathered, legal issues
counseled and policy adopted
by the AMAA Board of
Directors, it became a
reality the presentation of
facts did not properly
present itself until now.
Can the Maine-Anjou breed
survive this battle?
Yes, without a doubt. Once
we get the commercial test
and breeders utilize common
sense in their breeding
schemes, over time the issue
will take care of itself.
The key to a quicker resolve
of this issue is open
communication and steady
stream of documented
information.
In closing the American
Maine-Anjou Association
realizes the potential harm
a genetic abnormality can
have on breeding program and
a breed. But with an
aggressive program of
research, policy and follow
up, the abnormality can be
controlled and eventually
eliminated. Please feel free
to give me a call with any
questions you may have on
this important issue.
March/April 2006
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